‘No virus that causes disease in humans has ever been known to mutate to change its mode of transmission.‘ Mainstream Media misinformation

In truth, no virus is fully modified or attenuated or killed during the vaccine manufacturing process. All vaccines, by their very nature, play off each other, generate a “synergistic” chain-reaction triggering further (more insidious) infections & disorders. In many cases the very signature disease/disorder they claim to protect you against is PRECISELY that which they inadvertently spread; albeit a more virulent “transforming” strain of the primary pathogen.

Essentially, ALL Viral vaccines become “weaponized” through the various stages of Vaccine development; further metastasizing in the vaccine host: ‘Mutations that can occur when the vaccine virus replicates in the body may result in more a virulent strain.‘

In practical terms, a synergy factor inevitably occurs when multiple ingredients such as heavy metals, live (attenuated/modified) viruses/or strands of DNA-RNA “heat treated virus”, antibiotic(s), formaldehyde, detergent(s), diploid cells (aborted fetal tissue), mycoplasma, phenol dye & excipient buffers are combined together in a vial mixture, generating a bio-active (electrical) chemical reaction – worsened by combinations with post-vaccination Prescription drugs; frequently seen as the tipping point which triggers/hastens an escalation of adverse neurological symptoms, a cascading degeneration (reduced Mitochondrial & Metabolic capacity) leading to varying degrees of auto-immune (multi-systemic) failure in the body.

‘Many live attenuated vaccines for animals (including humans) are manufactured by using cell lines from animals, which are known to produce infectious ‘endogenous retroviruses‘ (Remnants of ancestral exogenous retroviral infections fixed in the germline DNA); however, the risks of infection by ERVs from xenospecies through vaccination have been ignored.’ Laboratory of Signal Transduction, Department of Cell Biology, Institute for Virus Research, Kyoto University

‘When passaged in the laboratory (in either cell culture or lab animals), primary isolates often become attenuated. The attenuation is the result of adaptive genetic changes that the virus acquires in order to survive in its new environment. These genetic changes can be subtle (single nucleotide changes) or dramatic (genome deletions or rearrangements). Generally, the longer a virus is propagated in cell culture, or through non-natural animal hosts, the greater the attenuation. In fact, this is the basic methodology for the development of many live attenuated virus vaccines.’ Impact of Synthetic Genomics on the Threat of Bioterrorism with Viral Agents, Marc S. Collett/ViroDefense Inc.

Parents who received the first wave of (live) measles shots in 1963, and all those since, were subsequently stripped of their capacity to transfer natural immuno-protection to their baby, via the Placenta & Colostrum, due to the cross-contamination factor (hybrid measles virus now embedded in the mother/father’s germ line DNA – manifesting in a more virulent strain of measles, known as ‘Atypical Measles‘) generated by the shot, passed on to the new generation.

“I am very concerned that “immunologic memory” of adjuvant-containing vaccines is actually the basis of sensitization rather than the basis of immunity. Furthermore, I am very concerned that “successful” prevention of childhood diseases by means of short-term protective effects of live attenuated viral vaccines during childhood has led to the loss of maternal ability to transfer immuno-protection to their young, thereby leaving infants vulnerable to those diseases, should the exposure occur.

I am also very concerned that vaccination campaigns work by disrupting disease transmission, which reduces the chances of exposure, rather than by establishing a population’s immunity. By doing so, vaccination campaigns wipe out population’s immunity to childhood diseases rather than help to maintain it. If in prior decades there was naturally established herd immunity to childhood diseases among the adult population, then I am afraid that vaccination campaigns have ensured that it is long gone.

All of this is a direct outcome of the “desired” vaccination effects, the impact of which hasn’t been carefully thought through in advance of introducing mass vaccination. We thought that vaccines work just like natural immunity. Well, apparently they don’t and we are now reaping the consequences of that.

We would expect that vaccinated individuals would not be involved (or very minimally involved) in any outbreak of an infectious disease for which they have been vaccinated. Yet, when outbreaks are analyzed, it becomes apparent that most often this is not the case. Vaccinated individuals are indeed very frequently involved and constitute a high proportion of disease cases.

I think this is happening because vaccination does not engage the genuine mechanism of immunity. Vaccination typically engages the immune response—that is, everything that immunologists would theoretically “want” to see being engaged in the immune system. But apparently this is not enough to confer robust protection that matches natural immunity. Our knowledge of the immune system is far from being complete.” Dr Tetyana Obukhanych Ph.D

‘We found 18 reports of measles outbreaks in very highly immunized school populations where 71% to 99.8% of students were immunized against measles. Despite these high rates of immunization, 30% to 100% (mean, 77%) of all measles cases in these outbreaks occurred in previously immunized students. In our hypothetical school model, after more than 95% of schoolchildren are immunized against measles, the majority of measles cases occur in appropriately immunized children.

The apparent paradox is that as measles immunization rates rise to high levels in a population, measles becomes a disease of immunized persons. Because of the failure rate of the vaccine and the unique transmissibility of the measles virus, the currently available measles vaccine, used in a single-dose strategy, is unlikely to completely eliminate measles. The long-term success of a two-dose strategy to eliminate measles remains to be determined.‘ Poland GA, Jacobson RM., Department of Internal Medicine, Mayo Vaccine Research Group, Mayo Clinic and Foundation, Rochester, MN./1994

‘As one of many examples involving all infectious diseases of childhood against which vaccines have been developed, ever since any measles vaccines have been introduced and used in mass proportions, reports of outbreaks and epidemics of measles in even 100% vaccinated populations started filling pages in medical journals. It is less well known to the general public that vaccinated children started developing an especially vicious form of measles, due to the altered host immune response caused by the deleterious effect of the measles vaccines. It resisted all orthodox treatment and carried a high mortality rate. It has become known as atypical measles (AMS).

In the meantime, outbreaks of measles in vaccinated children have continued and intensified to this day. Contemporary observations of the ineffectiveness of vaccination indicate to me that the incidence of measles has increased and has not continued decreasing as it did for some 100 years before any type of measles vaccination was introduced.‘ Dr Viera Scheibner (PhD), International Medical Counsel on Vaccination

‘We developed case criteria on the basis of serology and rash distribution and morphology. In typical measles a maculopapular rash occurs first at the hairline, progresses caudally, is concentrated on the face and trunk, and is often accompanied by Koplik’s spots. In AMS the rash is morphologically a mixture of maculopapular, petechial, vesicular, and urticarial components. It usually begins and is concentrated primarily on the extremities, progresses cephalad, and is not accompanied by Koplik’s spots. Cases were classified as AMS if patients had 1) a rash with the distribution and morphology characteristic of AMS, and 2) a fourfold or greater rise in titer of complement-fixing measles antibody or a convalescent titer of 256.‘ Dr. E M Nichols M.D.

Note: ‘Measles-mumps-rubella (MMR) vaccines containing the Urabe strain of mumps were withdrawn in the United Kingdom in 1992 following demonstration of an increased risk of aseptic meningitis 15–35 days after vaccination.…A total of 894 children (in the UK) were admitted to a hospital in the second year of life with febrile convulsion, or convulsion not otherwise specified, between January 1998 and June 2002 and had a linked MMR vaccination record. These children had a total of 988 convulsion episodes (819 had one episode, 60 had two, 12 had three, two had four, and one had five).‘ American Journal of Epidemiology    

The MMR Vaccine has co-infected an entire generation of children with a more virulent strain of Measles, now referred to as Atypical measles (AMS). The Medical Industry has reluctantly confirmed this vaccine-derived mutation circulating throughout the environment:

‘Atypical measles occurred (occurs) in children who received formalin-inactivated (killed) measles vaccine that was in use in the United States from 1963 to 1968. These children developed high fever, a rash that was most prominent on the extremities and often included petechiae, and a high rate of pneumonitis. Recent studies in monkeys indicate that this illness was caused by antigen-antibody immune complexes resulting from incomplete maturation of the antibody response to the vaccine.‘ The Journal of Infectious Diseases

‘Atypical measles can occur in people who were immunized with a killed virus vaccine that was used from 1963-1967 and then exposed to the original virus. It can also occur in people who were immunized with the current vaccine but, for some reason, failed to develop immunity, and in people who are immunosuppressed.‘

Note: ‘The symptoms of atypical measles are different and more severe than the symptoms of typical measles.‘

Symptoms of Vaccine-derived hybrid strain of Measles (Atypical Measles/AMS):

1. Maculopapular ‘characterised by flat spots (macules) and tiny bumps (papules) on the surface of a tissue—usually understood to mean the skin or an organ (e.g., the liver or spleen).’  

2. Petechial ‘pertaining to tiny red or purple spots caused by an extravasation of blood into the skin.’

3. Vesicular ‘composed of or relating to small, saclike bodies.’

4. Urticarial ‘hives; a vascular reaction of the upper dermis marked by transient appearance of slightly elevated patches (wheals) which are redder or paler than the surrounding skin and often attended by severe itching.’

‘Some vaccines, including the MMR, smallpox, and chickenpox vaccines, contain live viruses. By giving three and sometimes four live viruses together, the risk of developing a lifetime viral infection (a persistent viral infection) increases tremendously. This is especially so with the MMR vaccine, which contains two live viruses known to suppress the immune system for months.‘ Dr. Russell L. Blaylock M.D. 

‘An outbreak of measles occurred in a high school with a documented vaccination level of 98 per cent. Nineteen (70 per cent) of the cases were students who had histories of measles vaccination at 12 months of age or older and are therefore considered vaccine failures…Vaccine failures among apparently adequately vaccinated individuals were sources of infection for at least 48 per cent of the cases in the outbreak.‘ Measles outbreak in a vaccinated school population: epidemiology, chains of transmission and the role of vaccine failures – American Journal of Public Health 05/1987

Note: ‘Vaccine failures among apparently adequately vaccinated individuals were sources of infection for at least 48 per cent of the cases in the outbreak.’

‘Vaccination coverage for the total population was 99.0%. Incomplete vaccination coverage is not a valid explanation for the Quebec City measles outbreak.‘ Major measles epidemic in the region of Quebec (1989) despite a 99% vaccine coverage – Boulianne N1, De Serres G, Duval B, Joly JR, Meyer F, Déry P, Alary M, Le Hénaff D, Thériault N./PubMed 1991

‘The largest measles epidemic in North America in the last decade, occurred in 2011 in Quebec, Canada, where rates of 1- and 2-dose vaccine coverage among children 3 years of age were 95%-97% and 90%, respectively, with 3%-5% unvaccinated.‘ Largest measles epidemic in North America in a decade–Quebec, Canada, 2011: contribution of susceptibility, serendipity, and superspreading events: De Serres G1, Markowski F, Toth E, Landry M, Auger D, Mercier M, Bélanger P, Turmel B, Arruda H, Boulianne N, Ward BJ, Skowronski DM./PubMed

Dr. Andrew Wakefield’s 1998 Study demonstrated ‘anti-myelin antibodies (demyelination) and digestive tract pathologies‘ in children with autism after being given the Urabe strain triple live virus MMR vaccine. All 12 children in the Study had intestinal abnormalities (known as Inflammatory Bowel Disease), with chronic inflammation in the colon in 11 of the children.

Noted behavioral disorders included autism in 9, disintegrative psychosis in 1, and possible post-viral or vaccinal encephalitis (acute brain inflammation) in 2.

Since that release there have been countless other studies verifying exactly the same pattern – including the presence of measles lingering in the bowels of young children who have gotten of the Measles-Mumps-Rubella shot (MMR).

The Urabe strain MMR vaccine purchased by GlaxoSmitheKline for distribution in the UK, a triple live virus version, was directly responsible for an sudden spike in childhood Meningitis in the UK. It had been banned from use in Canada & was warned against further use in Britain by Canadian specialists. This was all suppressed by GSK & The British Gov’t. They are solely to blame for this crisis.

‘‘The (UK) Government waited another two years before it decided to stop using Urabe MMR in 1992, after the manufacturers told officials that they would stop making it. It was replaced with MMR II, which has a different mumps component.

Prof Kent Woods, chief executive officer of the Medicines and Healthcare products Regulatory Agency, confirmed that the UK authorities had been aware of “sporadic cases” in Canada. However, the risk of meningoencephalitis from Urabe MMR was lower than the risk of the same condition resulting from “wild-type mumps virus”, he said.

Urabe MMR was withdrawn “following reports of generally mild transient meningitis caused by the mumps vaccine virus in some children who recently received the Urabe mumps vaccine containing products.‘ The Telegraph/2007

‘The Urabe AM9 vaccine was developed in Japan and was originally attenuated by six passages of a wild isolate in chicken amniotic cavity, after which the virus underwent two plaque purifications in quail fibroblasts.

Specifically, the Urabe AM9 component of trivalent measles, mumps and rubella vaccines was associated with meningitis (inflammation of the meninges, the thin, membranous covering of the brain and the spinal cord)and parotitis (inflammation and infection of the salivary glands).

“I advocated very strongly for the use of the single vaccines, single Measles, Mumps, Rubella, which were available at the time in the UK. The vaccination uptake did not fall. MMR went down but there was a reciprocal increase in the use of single vaccines. Measles did not come back. Then, 6 months after I made that recommendation, the Gov’t in the UK unilaterally withdrew importation license for the single vaccines; thereby depriving parents of an opportunity to vaccinate their children. THEN, vaccination rates went down, THEN measles came back. The British Gov’t is solely responsible for that happening.

I have also found that regressive behavioral disorder (RBD) in children is associated with measles, mumps and rubella (MMR) vaccination. These children all have gastrointestinal symptoms including abdominal pain, diarrhea, and in some cases food intolerance. It is significant that this syndrome only appeared with the introduction of the polyvalent MMR vaccine in 1988 rather than with the monovalent measles vaccine introduced in 1968. This indicates that MMR is responsible for this condition rather than just the measles virus and that accordingly a transfer factor specific for the components other than the measles virus in MMR may be required.” Dr. Andrew Wakefield

A mutagenic Measles viral strain (atypical measles) commonly infects the bowels/intestines of children with Autism, manifesting as Inflammatory Bowel Disease – Intermediate/Advanced Crohns & Colitis. Merck’s official package insert for the Measles, Mumps, Rubella vaccine explains why: ‘M-M-R II has not been evaluated for carcinogenic or mutagenic potential, or potential to impair fertility.’

Measles, Mumps, Rubella Vaccine (series) – 1st round given at 12-15 months old: ‘The evidence convincingly supports a causal relationship between MMR vaccine and measles inclusion body enchephalitis (inflammation of the Brain & Meninges/Meningoencephalitis manifesting as ‘diffuse and/or focal neuropsychological dysfunction‘ – reaction to synergistic heavy metal-excipient accumulation of ’sludge’ toxicity) in individuals with demonstrated immune deficiencies.’ P.110 Institute of Medicine Report on Adverse Effects of Vaccines/2011

Official Package Insert: ‘M-M-R II is a sterile lyophilized preparation of (1) ATTENUVAX* (Measles Virus Vaccine Live), a more attenuated line of measles virus, derived from Enders’ attenuated Edmonston strain and propagated in chick embryo cell culture; (2) MUMPSVAX* (Mumps Virus Vaccine Live), the Jeryl Lynn** (B level) strain of mumps virus propagated in chick embryo cell culture; and (3) MERUVAX* II (Rubella Virus Vaccine Live), the Wistar RA 27/3 strain of live attenuated rubella virus propagated in WI-38 human diploid lung fibroblasts.’

‘Measles vaccine can cause problems (e.g. fatal giant cell pneumonia) in those  with severely compromised cell-mediated immunity. If a patient has an impaired cell-mediated immune response, there is  continued growth of the virus in the lungs leading to giant cell  pneumonia (such patients may not have a rash). This is rare, but often  fatal.’ Dr. Margaret Hunt, University of South Carolina School of Medicine

‘The development of giant cells has been illustrated in tissue cultures  infected with adenoviruses and measles viruses, and in ferrets infected  with distemper viruses.‘ Giant Cell Pneumonia: Clinicopathologic and  Experimental Studies, J. M. Adams,, D. T. Imagawa,, Miye Yoshimori, R.  W. Huntington 

‘One study, in which autopsied elderly were examined for the presence of the measles virus, found that 20% of the brains had live measles viruses and 45% of other organs were infected. These viruses were highly mutated, meaning that they could be just as potent as other measles viruses, but could be even more virulent. Worse, is that in most cases they cause a smoldering destruction of tissues without the obvious symptoms of infection, which has been shown in a number of studies.’ Dr. Russell L. Blaylock M.D.

Government data admits to a probable causal link between vaccines & the (inexplicable) presence of Measles in the bowels of young children; while promoting a deliberate cover-up of these inherent risks to the general public,

‘That Crohn’s disease and other chronic inflammatory illnesses of the intestine might be caused by a virus such as measles is an interesting hypothesis. Until the present time, microbiologic and epidemiologic arguments either for or against this hypothesis have not been very convincing. It is not very likely that other epidemiologic studies will provide conclusive evidence. In fact, it would be difficult to find a population that includes both individuals who have been exposed to the virus or to the vaccine and individuals who have not been exposed. However, new microbiologic studies might prove conclusive.

First, it would be necessary to demonstrate that the measles virus is indeed present in the lesions, that it is active, and that it contributes to inflammatory responses. Also, it would be necessary to prove that the pathogenic reaction can be induced by the wild virus and by the attenuated viruses present in vaccines. Strains and attenuation procedures vary from one manufacturer to another, and it is far from certain that all strains have the same ability to persist in tissues and to subsequently produce chronic inflammations.

As was stated above, measles vaccine does not seem to be associated with SSPE (subacute sclerosing panencephalitis), although the wild virus may be isolated (with difficulty) in patients with SSPE. The measles virus was isolated neither in patients with Crohn’s disease or other chronic inflammatory diseases (Paget’s disease, active chronic hepatitis, multiple sclerosis) in which a role for it has been claimed on morphologic, histologic or serologic grounds.

Current scientific data do not permit a causal link to be drawn between the measles virus and chronic inflammatory bowel diseases. While awaiting production and publication of other research, it would not be appropriate to alarm recipients of the vaccine by notifying them of this hypothetical risk, thus jeopardizing an immunization program of proven benefit.’ Identification of Genetic Mutations Associated With Attenuation and Changes in Tropism of Urabe Mumps Virus, Journal of Medical Virology 81:130–138/2009 

Note:  ‘…it would not be appropriate to alarm recipients of the vaccine by notifying them of this hypothetical risk, thus jeopardizing an immunization program of proven (cost) benefit.’

Vaccinated individuals are highly susceptible to viral shedding for weeks, months, and often years following the inoculation procedure. Here is a recent Peer-reviewed analysis of an individual who received a dose of the Oral Polio Vaccine (OPV) in 1986, and has subsequently been virally shedding a mutated, more virulent (weaponized) strain of Polio for 28 years.

‘The latest isolate available was from 4th March 2015 showing a 17.7% VP1 sequence drift from Sabin 2 poliovirus. Phylogenetic analyses in the capsid region confirmed that the iVDPV strains were genetically related, sequentially evolved from Sabin 2 and distinct from other type 2 VDPVs and wild polioviruses  

The date of the initiating OPV dose was estimated to be 11th March 1986 [HPD95 = 6th July 1983-11th January 1989], relatively close to 4th August 1986, the date of the patient’s last known OPV vaccination. It is therefore most likely that this individual has been excreting (virally shedding a mutagenic/chemically synthesized strain of) poliovirus for around 28 years.

Our results highlight the need for improving the standardisation of sIPV products in terms of measuring vaccine potency and defining the protective human dose.‘ Twenty-Eight Years of Poliovirus Replication in an Immunodeficient Individual: Impact on the Global Polio Eradication Initiative – Dr.s Glynis Dunn, Dimitra Klapsa, Thomas Wilton, Lindsay Stone, Philip D. Minor, Javier Martin, National Institute for Biological Standards and Contol, Hertfordshire, Public Library of Science Pathogens/August 27, 2015